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For instance, human SET7/9 has just lately been reported to methylate Lys189 in the normal transcription factor TAF10, resulting in an increased affinity for RNA polymerase II and transcriptional activation of certain TAF10-dependent genes 37 SET7/9 has also been reported to methylate p53, rising the stability of this quick lived tumor-suppressor protein 38 These observations suggest that we should not narrowly define the SET-domain proteins as histone lysine methyltransferases however instead name them protein lysine methyltransferases.
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